Sentence examples for genes we hypothesized from inspiring English sources

If clone 5F7 indeed regulates the OLIG genes, we hypothesized that they may interact in chromosomes.

Based on the analysis of target genes, we hypothesized that miRNAs regulate development through a particular emphasis on complex stages rather than general regulatory mechanisms.

Based on the overrepresentation of E-box-containing transcription factor binding sites among rapamycin-regulated genes, we hypothesized that c-Myc plays a role in the regulation of gene expression by mTOR.

Since PPARγ-null TS cells preferentially and prematurely differentiate into TGC at the expense of labyrinthine cell types, and since PPARγ activation by rosiglitazone downregulates markers of TGC while upregulating some labyrinthine genes, we hypothesized that PPARγ promotes labyrinthine/syncytiotrophoblastic differentiation.

If CENP-A is indeed co-opting accessibility pathways to accumulate at genes, we hypothesized that chromatin accessibility might play a role in ectopic CENP-A localization.

As non-HLA genes, we hypothesized that NOTCH4, TNXB, BTNL2, and C6orf10 all would be good candidates for further clinical and laboratory studies.

As the expression of XIK1 is induced as early as within 2 h post Xoo inoculation and reduced expression of XIK1 compromises the expression of PTI-related genes, we hypothesize that XIK1 regulates the early events of XA21-mediated signaling.

Considering the close evolutionary relationship among SMYD1, SMYD2 and SMYD3 genes, we hypothesize that they may share some common ancient mechanisms.

We therefore undertook a study of the 16p11.2 region to investigate the role of genetic variation in eight candidate genes we hypothesize may represent risk loci for autism spectrum disorders.

Because L1 insertions in these regions are generally more than 20 kb away from genes, we hypothesize that negative selection pressure against deleterious effects of L1 insertion could also play a role in the genomic distribution of L1 elements.

Based on these genes, we hypothesize that the basis of the tolerance at the cellular level is probably through mitigation of the oxidative burst and osmotic adjustment.