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This hypothesis is also consistent with the observation of no DOMO in the Group 1 Type I S genes lacking the repeats.
This enzyme is encoded within a mobile group I intron, located within the 25S coding sequences of Physarum polycephalum, that can spread to rRNA genes lacking the intron as a result of a gene conversion mechanism initiated by I-PpoI cleavage [30].
In order to determine if any of the SigT-dependent genes lacking the conserved SigT promoter binding motif were regulated by a pathway involving SigU, we used microarrays to compare the carbon starvation-induced expression levels in a wild type strain (LS101) and a sigU null mutant strain (LS3547).
Of 508 genes lacking the motifs, 40 (7.9%) were detected as cA genes.
Similarly, genes lacking the H3K4me3 mark were significantly more tissue-specific than all expressed genes in DSX (P < 2.2 × 10−16; Figure 3c).
It was reported in Arabidopsis thaliana that H3K4me3-modified genes tend to show less tissue-specificity compared to genes lacking the mark [ 27, 28], a trend we confirmed in Eucalyptus.
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All four human DUXY genes lack the potential to encode the complete second homeodomain [8].
Indeed, many commensal orthologs of phase-variable genes lack the repeat elements that participate in phase variation.
We also examined μ expression in 14 independent cell lines in which the reporter genes lacked the q'r's' segment (MEP', PEP', EP'), and found that these cell lines uniformly failed to express the μ gene at a detectable level.
The truncated proteins encoded by these genes lack the 26 first amino acids from the N-terminal domain (LdTopIΔL from now on) and three increasing length segments from the C-terminal end called LdTopIΔ467L, LdTopIΔ490L and LdTopIΔ519L.
All trnS genes lack the DHU stem.
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