These genes include the easily-shocked gene encoding an ethanolamine kinase; the slamdance gene encoding an aminopeptidase; and the kazachoc (kcc) gene encoding a potassium-chloride co-transporter.
These genes include the TNF family member eiger and the novel immunity gene edin (elevated during infection; synonym CG32185), both of which are more strongly induced by infection of wntD mutants compared to controls.
Examples of risk genes include the two lysosomal enzyme coding genes GBA and SMPD1, which are associated with a 5-fold and 9-fold increased risk of PD, respectively.
The SREBP-target genes include the important lipogenic and cholesterogenic genes such as fatty acid synthase (Fasn) and HMG-CoA synthase (Hmgcr) (Amemiya-Kudo et al. 2002).
These genes include the coding regions for 12S and 16S rRNA, 22 tRNAs, and the 13 hydrophobic proteins of the electron transport chain [1].
These genes include the pst operon (pstS, pstC, pstA, pstBA, pstBB), a gene required for cytochrome bd production (cydA), and several genes encoding hypothetical proteins (yddT, ygxB, yrhE, yusU, and ywtG).
Spliced HAC1 RNA is translated into a functional transcription factor, which activates the expression of UPR target genes include the folding chaperones KAR2 and PDI1 (Mattanovich et al. 2004).
Other examples of heritability in evolution that are not under the direct control of genes include the inheritance of cultural traits and symbiogenesis.
The genes include the initial selected signature genes and those added through the iterative enrichment analysis.
Such genes include the previously mentioned BMP2, CD44, β1 integrin subunit, FGF2, FGFR2 and PTHLP (Table 1).
These EBV genes include the latent membrane proteins (LMP 1 and LMP2a as well as the EBV nuclear antigens (EBNA 2, and EBNA1, among others.
