These findings, shown in Figure 4, were validated with considering hub genes in three centrality contexts: degree, betweenness and closeness.
The primers were designed based on dual priming oligonucleotide (DPO) technology to detect all of the 19 SAg genes in three sets of PCR.
To examine the constitutive and developmental expression of OsGLP genes, expression was monitored using gene-specific primers for all 12 genes in three positions of non-stressed leaves in three cultivars: Azucena, IR64, and SHZ-2 (Table 4).
Following the process, p16, E-cadherin, and p15 ssDNA-GNP probe with three domains were constructed and utilized to assay the methylation of these tumor suppressor genes in three cancer cell lines.
(E) LM2 cells or mouse breast cancer 4T1 cells were treated with 1 µmol/L Ponatinib at 24 h, heat map representing the expression of 6 metastatic-related genes in three independent experiments.
We performed a genetic association study of serotonin metabolism-related genes in three population groups: one affected with MOH, one with episodic migraine without aura and without drug abuse (MO) and one control population without headache and without drug abuse.
A total of 119 unique homologous genes (416 genes in three genomes) occupied 150 possible ranks.
We prioritized all genes in three GBS reference genomes to find potential virulence genes that are yet to be recognized.
Additionally, we selected four genes in three of the GSEA categories for validation by qPCR at P13 (Fig. 5C).
The four types of substitutions related to DNA methylation could have affected protein products of the genes in three approaches.
Initially, we sequenced all coding exons of the TARDBP, MTHFR, and MFN2 genes in three affected and five control animals.
