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Both sex-related and sex loci harbor TPT, HMG, and RNA helicase genes forming a syntenic gene cluster.
The six known APOL genes are exclusively present in primates, with the APOL1-4 genes forming a tight cluster on human chromosome 22q13.1 [30].
Moreover, as processed pseudogenes evolve much faster than functional genes, it is less likely for them to cluster with their parental genes forming a topology strictly like Figure 3A except when they were born very recently.
Previous nomenclature classified the Mups into three groups, identifying an unknown number of highly similar Mup genes to comprise one group, potential pseudogenes in a second group, and more divergent Mup genes forming a third group [17], [19].
The database contained temporal expression profiles of 5068 genes forming a 5068 × 9 data matrix.
The same principle can be applied to the disease genes forming a disease gene network.
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The mouse class I OR gene family consists of 158 genes, forming an approximately 3 Mb gene cluster [ 20, 21].
Genes form a biological blueprint that is largely reactive in nature, and the TCF7L2 gene was apparently quiescent during most of our evolution.
The resulting genes form a sequence.
The blocked 735 reactions linked to 533 genes, formed a platform for guided gene deletions in B. subtilis to generate simplified host strains.
Indeed the autotrophic arsenite oxidase genes form a distinct phylogenetic group, separated from previously described heterotrophic arsenite oxidase genes, with the exception of the heterotroph Agrobacterium tumefaciens.
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