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Animals defective in both SynMuv A and SynMuv B genes display a multivulva (Muv) phenotype.
Animals defective in both SynMuv A and SynMuv B genes display a multi-vulva (Muv) phenotype.
On the one hand, Prom+ genes display a higher level of expression than a random reference.
Cancer cells that have acquired mutations in either oncogenes or tumor suppressor genes display a characteristic uncontrolled proliferation phenotype [20].
Most genes display a value of p1-p2 close to zero, indicating a lack of significant imprinting.
Significantly, these unresponsive genes display a similar likelihood of requirement for growth fitness under H2O2-induced stress condition as the bound-induced genes (Figure 5F).
Cod+ genes display a lower than expected level of expression, and their centrality is not correlated with their level of expression.
Such genes display a modest increase of TR which is not paralleled by experimentally determined RA values, pointing to a sustained mRNA destabilization (Fig. 7).
Considering that many genes display a nonrandom usage of synonymous codons for specific amino acids, the SWAAP 1.0.3 software (http://www.bacteriamuseum.org/SWAAP/SwaapPage.htm) was used to compute the codon frequency and the relative synonymous codon usage [46] for all genotypes.
In summary, intestinal gene expression shows major changes by increasing the dietary fat content in mouse diets and numerous genes display a dose-dependent alteration in mRNA levels depending on dietary fat content.
At least four of the dominant peroxin genes display a slightly increased mRNA expression level in pept-1 when compared to wild type (prx-5 (+1.25), prx-6 (+1.1), prx-11 (+1.83) and prx-13 (+1.31)).
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