In C. elegans, components of this pathway are also known as synMuv B genes because they exhibit a synthetic (syn) multi-vulva (Muv) phenotype when combined with a mutation in a separate synMuv A class of genes.
Population geneticists usually prefer to study what are called neutral genes, because they accumulate regular random changes in their DNA, which serve as a useful genetic clock.
Ambry Genetics already offers a test for 14 genes that raise the risk of breast cancer, but it leaves out the two most important genes because they are patented by Myriad.
Tissue-specific genes are longer than housekeeping genes because they have more functional domains, which is an indication of their more-complex protein architecture.
And many of the younger firms, he argues, want fewer legal fences around genes because they are trying to create and market whole-genome technologies that use thousands of genes.
We chose these two genes because they showed high and moderate up-regulation under LD respectively.
We ignored those possible retropositions between intronless genes because they might not be generated by retroposition.
We examined the expression levels of these genes, because they have a role in antifungal defense [48].
These control genes, also called normalizers or reference genes, are often chosen from "housekeeping" genes because they are expected to be evenly expressed across most tissues and samples.
We chose repressed genes because they are down regulated upon entry into dauer (when DAF-16 is active) and RNAi also represses expression.
The cpr-1, F52E1.5 and clc-1 genes are also predicted to be protective genes because they are induced upon infection [38], [39], [56].
