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Due to the complexity and increasing decentralisation of the energy infrastructure, as well as growing penetration of renewable generation and proliferation of energy prosumers, the way in which energy consumption in buildings is managed must change.
The presence of CD34/CD68/DLK cells grouped in spheres suggest that paracrine interactions between these cell types plays an important role in the generation and proliferation of new preadipocytes.
In accordance to these findings, Yoshida et al [33] have recently shown that mild hypoxia is the optimal condition for both the generation and proliferation of induced pluripotent stem cells (iPS) from human somatic cells in comparison with 1% or 21% O2, leading respectively to cytotoxic effects or to a less efficient reprogramming process.
The induction phase occurs in the early stages of the T cell immunity, initiating tumor-antigen recognition and antigen specific T cell generation and proliferation, while the effector phase occurs in later stages of the immune response, affecting T cell activation, recognition and destruction of antigen-expressing tumor cells.
Therefore, it is fundamentally important to understand how the generation and proliferation of INPs are regulated.
These results indicate that changes to NOX1 expression probably specifically account for the effect of PGC-1α on ROS generation and proliferation.
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Although the vast majority of such recombinants most likely never survive for long enough to be detected, the occasional generation, emergence and proliferation of a new, and presumably highly-fit, recombinant variant from such mixed infections can have extremely long-term negative consequences for cassava farmers.
The CSE-specific miRNAs significantly inhibited CSE expression and H2S production, increased reactive oxygen species generation, and induced proliferation of human aorta smooth muscle cells (HASMCs).
Collectively, these data suggest that NOX1 activation may underlie, at least in part, the enhanced effect of PGC-1α knockdown on ROS generation and cell proliferation.
The phosphorylation of FGFR4 enhanced the production of metalloproteinase (MMP -7 and MMP -7n-andivated protein kinase kinase (Mek)/extracellular signal-regulated kinase (Erk), which was accompanied by excessive vessel generation and cell proteinration.
NS-398, a COX-2-selective inhibitor, suppressed PGE2 generation and cell proliferation in mock cells by ~60%and~25%5% respectively, whereas no further decreases in PGE2 production and cell growth were found in mPGES-1-KD cells.
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