Exact(9)
The cooperation between p53 and NF-kappaB may play a physiological role to neutralize apoptosis in response to DNA damage generated during inflammation.
Collectively, these results suggest that sulfation by SULT1A3 in lung endothelial and epithelial cells may play a role in the inactivation and/or disposal of excess chlorotyrosine and nitrotyrosine generated during inflammation.
A large amount of NO is generated during inflammation (review in [ 23]).
Larry Marnett (Vanderbilt) focused on the application of mass spectrometry for profiling electrophile conjugates that are generated during inflammation.
It is now established that thiosulfate can be converted to tetrathionate upon reaction with reactive oxygen species that are generated during inflammation [ 51].
For example, reactive oxygen species generated during inflammation react with endogenous, luminal sulfur compounds (thiosulphate) to form a new respiratory electron acceptor, tetrathionate, which S. typhimurium has evolved to use, while commensals lack the required metabolic pathways.
Similar(51)
For example low MH HA fragments generated during joint inflammation may promote chemokine gene expression by macrophages.
Protease-activated receptor-1 (PAR-1) is expressed by multiple cell types present in the lungs and can be activated by various proteases generated during acute inflammation.
Moreover, as suggested by De Marzo et al (1999b), the genomic DNA damage generated during chronic inflammation induced by bacterial insult may enhance the likelihood of neoplastic transformation.
In contrast, Th17 cells generated during acute inflammation against Candida albicans maintained their signature cytokine expression, most of which were eYFP+IL-17A+.
Some researchers have reported that VE is directly involved in the maintenance of the balance of oxidative reactions generated during the inflammation [ 17– 20].
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