We generated a mouse encoding p53 lacking the proline-rich domain (p53ΔP).
We recently generated a mouse with a knock-down in FKRP in the muscle but not the central nervous system (FKRPMD).
Through concomitant Cre-mediated inactivation of E-cadherin and PTEN in mammary epithelium, we generated a mouse model of classical ILC (CLC), the main histological ILC subtype.
We generated a mouse model lacking both collagen IX and COMP to study the potential complementary role of these proteins in skeletal development.
Recently, we generated a mouse model with ectopic integration of full-length hPARP-1 [Mangerich, A., Scherthan, H., Diefenbach, J., Kloz, U., van der Hoeven, F., Beneke, S. and Bürkle, A., 2009.
Therefore, Wang et al. have generated a mouse model of AC3 deficiency to test this hypothesis.
Investigators have also generated a mouse model with MBEN by simultaneously overexpressing Shh and HGF [22].
To study the role of Sei1, we have generated a mouse line deficient for this gene.
We have generated a mouse with a targeted deletion of the Klrd1 gene encoding CD94.
In this study we generated a mouse line that expresses functional CRE recombinase from the native melanopsin locus.
To test this idea, we generated a mouse transgenic line that expresses SMAD1 upon Cre-induced recombination (Figure S5).
