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After making model of gastric functional disorder (FD), part of model mice were injected intravenously (i.v).v
Hereby, we concluded that the secretion of pepsinogen in administration group could be increased slightly because of gastric functional disorder has been improved via injection with oMWCNTs.
It is interesting to speculate whether this group could be identified by tonometry, perhaps in combination with a test of gastric functional response.
As MRI is becoming more and more widely available, involves no exposure to ionizing radiation and is accepted by patients in the clinical setting since it does not require any oral intubation, MRI has been proposed for evaluation of gastric functional disorders.
Fourth, the current study only discussed two mucins (i.e. MUC1 and MUC2) and one pepsinogen PGC without inclusion of other mucins like MUC4 which has been reported increased expression in different types of gastric cancer like adenocarcinoma and SRCC [ 35] or other gastric functional proteins like pepsinogen A [ 36] and trefoil factors family [ 37- 40].
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Three experimental models of gastric function were investigated; highly-purified functional H,K-ATPase (proton pump) isolated from the gastric mucosa of slaughterhouse hogs, isolated rabbit gastric parietal cells, and human gastric adenocarcinoma cells in culture.
Key words that were used included: functional tests, gastric emptying, gastric motility, gastric physiology and MRI.
A systematic search for relevant original publications was conducted on MEDLINE from 1990 to 2008 using the following search keys: functional tests, gastric physiology, gastric emptying, gastric motility and MRI.
7 8 Patterns of hospitalisation of preterm infants are also associated with the presence of clinical symptoms of abnormal gastrointestinal function such as vomiting, regurgitation, gastric residuals and functional constipation.
Mice deficient in the H+,K+ ATPase β-subunit (HKβ) do not have a functional gastric H+,K+ ATPase, are unable to secrete gastric acid and show prominent morphological changes in their parietal cells, including a dramatic reduction in the size of the TVE (Scarff et al., 1999).
Such extensive medication may have caused physical or functional gastric mucosal damage that facilitated entry of the organisms into the gastric wall.
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