Since little is known regarding SETD6 functions, we set out to identify SETD6-associated proteins.
To better understand how mir-83 functions, we set out to determine its mRNA targets and miRNA coregulators in C. elegans.
To explore the role of the network in circadian function, we set out to perturb vesicle traffic as a way of disrupting intercellular communication.
To extend these studies, and investigate the epigenetic dependency of fly development on Tip60 HAT function, we set out to create a fly line producing GAL4 inducible dominant negative acting dTip60 proteins specifically defective in their catalytic HAT activity.
To gain clues to USP45 function, we set out to identify interacting proteins.
In order to establish the molecular basis of SYCP3 function, we set out to find a robust means for its recombinant production.
In a continued effort to understand the relationship of cardiac metabolism and cell function, we set out to test the effects of acetate on contraction.
Following the definition of criteria, prioritisation of markers and classification according to conclusiveness for clinical relevance and involvement of immune function, we set out to devise a framework for the interpretation of changes observed in the different types of immune function markers, taking into account the type of marker and the changes observed relative to a defined reference range.
Therefore, we additionally require that the degree of the polynomial be minimal, where the degree of some polynomial for almost all (m1, m2,..., m N ) ∈ ℕ N, is defined as For we define We now show that indeed satisfies the three requirements for interpolation functions that we set out at the beginning (see section on continuous homologues of Boolean functions).
To determine whether the Cap-H2 Mrg-binding motif is required for its compaction and antipairing functions, we first set out to determine whether the Mrg-binding motif is required for Cap-H2 mediated Cap-H2 mediatedpersal.
