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This provides evidence against previous claims that the rTPJ is not selective for social cognition (Mitchell 2008) and, therefore, adds to the ongoing theoretical discussions and increasing interest in the functioning of the "social brain" (Blakemore et al. 2004; Insel and Fernald 2004; Adolphs 2009; Blakemore 2012; Dunbar 2012).
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Nonetheless, our findings emphasize that irregularities in amygdala function need to be considered within the broader context of the social brain network.
Implications for underlying organization of the social brain are discussed.
The coordination of internal demands and social rules has been called the social brain function [1], [2].
These findings advance neural models that associate the structure and function of the human brain with social decision-making and the tendency trust other people.
But various technical difficulties have made the study of social brain function extremely difficult.
Myelination is also critical for normal, everyday functioning of the brain.
The alpha-synuclein protein, a Lewy body, prevents the normal functioning of the brain.
Others have raised the possibility that such drug use could induce unanticipated changes to the complex biosocial and psychological functions of the brain, altering our social behaviour in an altogether different way [ 81].
Since conventional methods could not handle such massive data, to date there has been almost no study of social brain function at the single-cell level.
Because experience drives cortical specialization, reduced attention to "others" results in a failure of specialization and less efficient function of the brain regions that mediate social cognition [ 54].
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