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To separate this functional phenomenon from a particular mechanism, we refer to it as specific immune priming.
However, this functional phenomenon seems not be correlated to any structural connectivity changes since both whole-brain and thalamic ROI DTI analyses have not demonstrated significant differences between VM, MwoA and HC.
Although this functional phenomenon is present in the absence of clinically relevant deficits, it may reflect a vulnerability to executive high-demanding conditions of daily living activities in patients with migraine.
These conditions were designed to mimic the tests performed in real-time and thereby provide a statistical evaluation of the functional phenomenon detected via our real-time exploration.
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Typical connectionist models, by contrast, follow a bottom-up approach, beginning with a characterization of neural mechanisms and exploring what macro-level functional phenomena might emerge.
On this view, there is nothing in the gap that should lead us to any bifurcation in the world between experiences and feelings on the one hand and physical or functional phenomena on the other.
The present study was designed to extend our previous results obtained in rd1 mouse (a relevant model of human recessive RP) to transgenic rhodopsin P23H mutant rat, a frequent mutation in dominant human disease and a good model for studying long-term effects and functional phenomena.
Although, different functional phenomena were explained in [ 1].
It is equally important to identify biochemical mechanisms of the observed functional phenomena.
In Systemic Functional Linguistics, Phenomena are optional participants.
Hence, the task of this explanatory reduction is to explain all functional biological phenomena via molecular biology.
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