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Endothelial function in peripheral conduit arteries reflects function in the human coronary circulation (12), and impaired endothelium-dependent vasodilation predicts cardiovascular events (13, 14).
Endothelial dysfunction is a marker of cardiovascular complications, and a close relation exists between endothelial function in the human coronary circulation and peripheral circulation (23, 24).
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These results demonstrate myogenic constriction in the human coronary microcirculation.
Cataloguing the geometry of the human coronary arteries: a potential tool for predicting risk of coronary artery disease.
With exclusion of the effect of I-R and elimination of the role of PGI2 and NO, we have demonstrated that hyperkalemic solutions [ 17– 19] and clinically used crystalloid cardioplegia/organ preservation solutions such as ST [ 42] and UW solutions [ 43] impair EDHF-mediated function in porcine or human coronary arteries and veins.
Focuses on localization of cognitive function in the human brain.
We investigated the function of vascular smooth muscle CCR5 in human coronary artery and saphenous vein, vascular tissues susceptible to atherosclerosis, and vasospasm.
Recently it was put forward that CGRP may act on the amylin receptor in TG [85] as well as in human coronary arteries [86].
Fasting Glucose Variability in Young Adulthood and Cognitive Function in Middle Age: The Coronary Artery Risk Development in Young Adults (CARDIA) Study.
Recent reviews (20) have examined the cardiovascular benefits of incretin therapy including enhanced cardiac myocyte viability after ischemic injury, increased systolic function in preclinical models and humans, coronary arterial vasodilatation, improved endothelial function, increased sodium excretion, and protection of neural cells against hyperglycemic injury.
Similar results were obtained in human coronary VSMCs (Fig. 4A).
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