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The unicellular eukaryotic Saccharomyces cerevisiae has provided a formidable model to bridge the sequence to function gap.
The regulation of NF-κB by reduced Cx43 protein level could be caused by absence of Cx43 function (gap junctional communication) or absence of Cx43 interactions with other proteins, such as c-Src.
Therefore, comparisons across a range of evolutionary divergences have helped bridge the sequence to function gap, from pinpointing DNA sequence elements [5,6] through to identifying virulence factors [13].
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In this paper, we conduct a study to predict the daily wave heights in different geographical regions using sequential learning algorithms, namely the Minimal Resource Allocation Network (MRAN) and the Growing and Pruning Radial Basis Function (GAP-RBF) network.
The authors have used a sequential learning algorithm in a growing and pruning radial basis function (GAP-RBF) network to estimate the relationship between the mentioned features and the corresponding quality measure.
Consequently, maintaining Cx43 function at gap junctions, thereby preserving gap junction intercellular communication, has the potential to attenuate malignant transformation and metastatic progression [ 14, 16, 17].
For gap-detection responses, percent-gap functions (equivalent to percent correct functions, except with no-gap percent incorrect values included to anchor the function at gap length = 0 ms; i.e., no gap) were calculated for each session.
GAP-stimulated hydrolysis is plotted as a function of GAP concentration for wild type and switch II glutamine mutant Rabs.
In [11], exact penalty functions via regularized gap function for variational inequalities have also been given.
Synchronization mismatch discomfort threshold (in ms) as a function of gap (bezel) width.
Nanodot arrays control astrocyte-astrocyte interaction by regulating the function of gap junction proteins.
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