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NO plays an essential role in regulating airway function and in the pathophysiology of inflammatory airway diseases.
Nitric oxide (NO) may play an essential role in regulating airway function and in the pathophysiology of inflammatory airway diseases [ 1].
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Additionally, research efforts are needed concerning the mechanisms underlying K2P5.1 function and regulation in the pathophysiology of RA.
The biological "target" of the drug and its function in the pathophysiology of the disease should be known [ 11- 13].
Nevertheless, several observations may support the role of normal variation in HPA axis function in the pathophysiology of PMS.
Progressive loss of beta-cell function is important in the pathophysiology of type 2 DM and may be responsible for SU failure.
Chemoreflex function is important in the pathophysiology of heart failure.
It is of note that reduced NMDA receptor function is implicated in the pathophysiology of ASD, as well as schizophrenia (48– 50).
miRNAs have a function in cell proliferation, differentiation, metabolism, apoptosis, development, and aging and in the pathophysiology of many diseases, namely, in oncogenesis, cardiovascular, and neurological disorders [ 12– 15].
These two different phenotypes possess different functions in the pathophysiology of sepsis.
Pulmonary hypertension and poor myocardial function often play a role in the pathophysiology of pulmonary disease [ 1].
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