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Interestingly, osteoclasts from ADO II patients show a reduced total resorption pit area but the area of the individual pits was the same [18] implying that some of the ADO II osteoclasts are still fully competent of bone resorption while others are inactive.
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Neary identified three main categories of concern for students which are the fear of doing harm to patients, the sense of not belonging to the nursing team and of not being fully competent on registration [ 28] which are similar to what our students mentioned in the focus group discussions.
These findings support the hypothesis that the chaperone-mediated rescued Parkin(C289G) is fully competent for enhancing mitophagy of damaged mitochondria.
At lower doses (1 10 ng/ml) of EGF, EGFR is only partially tyrosine phosphorylated and not fully competent for stimulating Tyr-phosphorylation of Tom1L1, inducing interaction with Tom1L1, or downstream signalling leading to Akt1 activation.
These results indicate that SVs are fully competent for release in the absence of the C2A domain of UNC-13L.
Thus, we believe that patients with the ATG16L1*300A allele are likely to be fully competent for the homeostatic functions of autophagy, yet may exhibit altered responses to bacterial components and infection in the gut milieu, where antigen burden is high.
It is also remarkable that we detect no OM biogenesis defect in strains carrying waaL15, demonstrating that the Lpt system is fully competent for the transport and assembly of LPS* despite the addition of both unnatural sugars and peptide stems.
Thus, stricto sensu, platelets do not appear to be fully competent for phagocytosis and their trapping of infectious pathogens remains unsolved.
These observations showed that stably transduced cells were fully competent to appropriately undergo a course of chondrogenesis, thus indicating that retroviral transduction did not influence the course of chondrogenic differentiation in the cells.
These results imply that mutations at positions 313 and 314 that do not inhibit formation of stable channels are fully competent for protein translocation.
Here we report that the M. musculus enhancers are fully competent to drive transcription in the context of the G. gallus IGL locus, but are unable to support high-levels of SHM/GCV, neither individually nor in combination.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com