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Based on data obtained from the aforementioned experiments, we conclude that P2X7R expressed on PDLSCs plays a positive role in PDLSC osteogenic differentiation under inflammatory conditions.
From the aforementioned experiments, there is an obvious implication that reduced regional perfusion in hypoxia equates to a hypoxic vasoconstriction although this has not been demonstrated directly in these studies.
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Prior to the measurement, the aqueous LS or HS solution from the aforementioned experiment was centrifuged for 10 min at 6000 rpm to remove impurities.
In the aforementioned experiments, we isolated CD34+/− cells from pooled hindlimb, pectoralis and triceps muscles.
The aforementioned experiments suggested that MyoD-lineage cells from the muscle migrated and repaired open fractures in the absence of other osteocompetent cells.
The aforementioned experiments demonstrated that the channel-based size exclusion is the major transport mechanism for our composite membranes.
All the aforementioned experiments were done by circular polarization of laser pulses.
In the aforementioned experiments, the number of fibres within each muscle biopsy was quantified.
The aforementioned experiment can be understood as a baseline for matching under non-ideal conditions.
The aforementioned experiment revealed a direct inhibition of IFN-β on the production of IFN-γ and IL-17.
Moreover, the aforementioned experiment on Wistar rats was terminated at the age of 110 weeks.
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CEO of Professional Science Editing for Scientists @ prosciediting.com