The frequency of odor-induced spike firing from individual sensory neurons was also reduced by pertussis toxin.
In addition, the subjects from Experiments 1 and 2 were pooled, and the frequencies of odor production and perception were calculated, [(number of subjects who failed to produce the odor/total number of subjects in Experiments 1 and 2) × 100].
This magnitude of effect could have public health importance because of the frequency and duration of odor episodes near CAFOs.
Inhibition of Go signaling lowered the odor-induced frequency of ab1A spikes and odor-evoked EAG response by an equivalent amount, i.e., 40-45% reduction in response.
The frequency distributions indicate the percentage of odor stimuli that excite a particular number of neurons.
Loss of Go signaling in ORNs reduced the amplitude and enhanced the termination of EAG responses, and decreased odor-induced spike frequency in individual ORNs independent of odor type or concentration.
Rhythmic changes in spike amplitude were observed in-phase with the rhythms of odor-evoked EAG responses, but spontaneous or odor-evoked spike frequency of olfactory neurons did not change [70].
Retrospective symptom prevalence data, collected from over 2000 adult respondents living near three different hazardous waste sites, were analyzed with respect to both self-reported "environmental worry" and frequency of perceiving environmental (particularly petrochemical) odors.
Although the structured reporting of odor by neighbors of swine CAFOs is a strength of our study, the frequency, duration, and intensity of reported hog odor episodes must be interpreted in the context of participants' daily activity patterns.
Odor-evoked LFP data were collected during the conditioning and test sessions and analyzed off-line using Spike 2. We performed power spectrum analysis on LFP data and compared the power spectra of five second pre-odor baseline periods to the five seconds of odor presentation in theta (5 10 Hz), beta (15 30 Hz), and high gamma (60 90 Hz) frequency bands.
