Sentence examples for fpi 1 from inspiring English sources

The Freiburg Personality Inventory (FPI-R) [ 24] includes the scales impaired well-being (FPI Factor SI), aggressive arousability (FPI Factor SII), poor satisfaction with life (FPI 1), arousability (FPI 5), emotional stress (FPI 7), physical complaints (FPI 8), health worry (FPI 9), and emotional instability (FPI N).

Cromakalim and NS1619 induced pial artery dilation was attenuated following FPI and MEAVP preadministration partially prevented such impairment (13±1 and 23±1, sham control; 2±1 and 5±1, FPI; and 9±1 and 15±2%, FPI-MEAVP pretreated for responses to cromakalim 10−8, 10−6 M, respectively).

Exposure of the cerebral cortex to a xanthine oxidase O−2 generating system blunted mastoparan induced pial artery dilation similar to FPI (10±1 and 17±1 vs. 2±1 and 3±1%).

Following FPI in the newborn, NMDA (10−8, 10−6 M) induced pial artery dilation was reversed to vasoconstriction and both NOC/oFQ and ET-1 receptor antagonists partially prevented such alterations (9 ± 1 and 16 ± 1, sham control; −7 ± 1 and −12 ± 1, FPI; −2 ± 1 and −3 ± 1, FPI-NOC/oFQ antagonist; and 2 ± 1 and 5 ± 1%, FPI-ET-1 antagonist).

One FPI (FP01), possessing a large aperture etalon (diameter: 116 mm), was installed at the EISCAT Tromsø site in 2009.

CSF NOC/oFQ was elevated within 60 min of FPI (70 ± 3 to 444 ± 51 pg/ml) but such release was attenuated by the ET-1 antagonist BQ123 in the newborn (66 ± 3 to 145 ± 10 pg/ml).

Only one FPI (FP01) uses the typical large-aperture etalon with a diameter of 116 mm and a tunable gap, giving higher sensitivity and more flexibility in the measurements.

Recently, the F. tularensis subspecies holarctica strain LVS Hfq protein was reported to play a role in virulence, possibly by negatively regulating the pdp operon of the FPI [13].

Insulin resistance was assessed using the homeostasis model assessment (HOMA-IR) originally described by Mathew et al. [30] HOMA-IR was calculated using the following equation: HOMA-IR (mg/dl × µU/ml) = FPG mg/dl × FPI µU/ml/405.

These include notably genes located in the Francisella pathogenicity island (FPI) [5], [6], [7], [8], [9], [10], [11], [12], and genes encoding the regulatory proteins MglA, SspA, FevR, PmrA and MigR, which regulate expression of the FPI [5], [13], [14], [15], [16], [17]; see [18] for a recent review.

Experimental and epidemiological studies in humans and animal models have demonstrated that predisposition to impaired glucose tolerance, blood pressure and coronary heart disease, are associated with either low or high FPI [9], [10], [11], [12], [13], [14], [23], [79], [80], [81].