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Otrf also was found to accelerate the expression of immune response gene such as IFN-γ against MDV infection [ 45].
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Tumor-promoters, such as phenobarbital, were found to accelerate and increase formation of preneoplastic islets.
Overexpressing Fyn was found to accelerate synapse loss and the onset of cognitive impairment in the J9 (APPswe/Ind) transgenic AD mouse model, while removing Fyn expression rescued synapse loss in the J20 (APPswe/Ind) transgenic AD model [ 64, 65].
NGF has been found to accelerate epithelial healing and induce migration of keratocytes [ 25, 26].
GmPti1 gene expression was found to accelerate in response to wounding and salicylic acid treatment in seedling leaves [ 8].
Enforced expression of A1-a in T-cells was found to accelerate T-cell survival in a1-a tg mice.
Rates of atrophy for the hippocampus have been found to accelerate during late life [29].
In addition, hypoxic conditions were found to accelerate Aurelia planula settlement and polyp survival in natural conditions [ 12], and Aurelia polyps and medusae were recently shown to react to hypoxia by increasing their expression of hif1α [ 48].
Hypoxia was found to accelerate this process.
Furthermore, inactivation of E2F was found to significantly accelerate tumor development in transgenic mice expressing Myc.
Reepithelialization was found to be accelerated after a single application of ESWT [ 12].
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