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This may be a reason that we found localisation at a summary statistics level: our more experienced participants could extract information more rapidly and therefore processed the images in greater detail.
We found localisation to be virtually entirely cytoplasmic, most probably mitochondrial, which in line with several previous observations in which hOGG1 immunohistochemical expression has been assessed (Salim et al, 2008; Ku et al, 2009).
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Finally, localisation of the EGFP-Irgm1 αK construct to the mycobacterial phagosome was reported while we found no localisation of the EGFP-Irgm1 αK construct to latex bead phagosomes.
No differences were found in localisation, stage or treatment of the tumours.
We found no localisation of citric acid cycle compounds in the mitochondrion such as fumarate and succinate which only occur in the cytosol.
We found that localisation of BMP-7 expression was unchanged in human hypertensive nephrosclerosis, but expression was reduced, as demonstrated by immunostaining and real-time PCR.
We found decreased localisation of CK1 ɛ and phosphorylated DVL2 (Ser143) at the signalling triaging centre centrosome after FZD7 knockdown in Stem-A OC cells.
Similarly, we found co-localisation of CCL5 and CD68 (figure 4D).
An increased risk was found for localisations of brain tumours other than the most exposed, e.g. temporal lobe, frontal lobe and cerebellum, which of course present a problem for drawing causal conclusions.
Some operators find the localisation of veins to be useful.
Interestingly, when analyzing the localisation of lesions in respect to brain areas in DM1 we found a main localisation in corresponding areas; central temporal and frontal areas, but also the insula region (data not shown).
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