Such visuovestibular cortical interaction could underlie the perceptual mechanisms that attempt to resolve visual motion from self-motion percepts while simultaneously optimizing visual form perception under natural conditions of vestibular activation such as during locomotion.
The occipito-temporal cortical activations were in the lateral occipital complex, a part of the ventral visual pathway active during visual form perception. Thus, macrospatial form perception preferentially recruits this region of extrastriate visual cortex, compared to microspatial form perception.
We then demonstrate the applicability of these principles to four specific domains of infant perception and/or cognition, (i.e., form perception, object unity, complex pattern perception, and understanding of causal events).
These major differences in the properties of cells at lower levels in each of the subdivisions led to the prediction that different visual functions, such as color, depth, movement, and form perception, should exhibit corresponding differences.
There is evidence that the neural processes underlying global form perception may be pattern specific.
Previous experimental evidence on the extent of V1 activity to global form perception is contradictory.
Visual risk factors include low-level visual difficulties such as reduced contrast perception [41] [44] and higher-level visual form perception deficits [45] [48].
If motion-defined form persistence reflects a transient motion-induced facilitation of static form perception that decays over time, one should be able to objectively measure the facilitation.
This allowed us to assess the relative importance of striate (V1) versus extrastriate cortex in global form perception. Stimuli were horizontal, rotational and radial Glass patterns.
Our experiments suggest that this information can interact with analyses of subsequent static input, resulting in a transient facilitation of static form perception.
