Sentence examples for formation highlights from inspiring English sources

The long postnatal persistence of CR cells in the hippocampal formation highlights their conventional roles as neurons integrating and relaying information to their prospective synaptic targets.

This link between cancer cell invasive ability and capacity for compact spheroid formation highlights the importance of targeting these multicellular aggregates.

The new physicochemical mechanism for kidney stone formation highlights the important role of phosphate, and the formation of calcium phosphate nidus.

Nevertheless, the use of a set of conserved proteins by morphologically diverse cell types to regulate cell cell contact formation highlights both the importance of the proteins for cellular function as well as their versatility that allows the regulation of similar aspects in different cell types.

The regions of high probability for coiled coil formation highlight localised differences between the species.

Interestingly, NAC-treatment reduced SARM-induced MPTP formation, highlighting that ROS production precedes loss of ΔΨm (Supplementary Figure S5b).

One of these genes, HECW2, plays an important role in ubiquitination, a prerequisite for chromatin remodelling and acrosome formation, highlighting the involvement of this gene in the knobbed acrosome defect and male infertility.

Some of these models resemble human HCCs and have contributed to varying degrees towards understanding the mechanisms and genes involved in HCC formation, highlighting the relevance of such mouse models.

The serial OCTs and videos supported anterior-posterior vitreofoveal traction as the initial event in macular hole formation, highlighted by case 2. Although case 1 had no evidence of vitreofoveolar traction and only a small defect in the ILM by OCT, the presence of a pre-foveolar operculum on clinical exam also supports this theory.

Finally, we could demonstrate that treatment of Apoe-deficient mice with a blocking antibody against CCL17 effectively reduced atherosclerotic plaque formation, highlighting the importance of CCL17 for Treg maintenance by DC in the context of vascular inflammation and identifying this chemokine as potential therapeutic target for the treatment of atherosclerosis (Weber et al, 2011).

Genome-wide analyses have identified deregulated miRNA expression in human malignancies [ 149] and a potential dual role in tumor formation, highlighting that miRNAs can modulate oncogenic or tumor suppressor pathways, including p53, c-MYC, RAS, and BCR-ABL, while expression of miRNAs themselves can be regulated by oncogenes or tumor suppressors.