Sentence examples for form of nitric from inspiring English sources

Most recently, genetically engineered mice with targeted disruption of the gene encoding the neuronal form of nitric oxide synthase have been studied.59 Hypertension of the LES was demonstrated as well as marked impairment of swallow-induced LES relaxation.

Animal models that include genetically engineered mice with targeted disruption of the gene encoding for the neuronal form of nitric oxide synthase and pharmacologic administration of inhibitors of nitric oxide substantiate the role of nitrergic denervation in achalasia.

We used G93A-mSOD1 mice to test the hypothesis that the degeneration of MNs is associated with an aberrant up-regulation of the inducible form of nitric oxide synthase (iNOS or NOS2) activity within MNs.

l-Arginine (l-Arg) is a conditionally essential amino acid for humans, which is the substrate for both arginase (ARG) and the inducible form of nitric oxide synthase (iNOS) enzymes.

In and near regions of urbanization and heavy industrial activity, the enhanced deposition and precipitation of sulfur in the form of sulfuric acid, and of nitrogen oxides in the form of nitric acid (HNO3), resulting from vehicular emissions, have been associated with damage to fish populations, forests, statues, and building exteriors.

Primary NO x emissions are mostly in the form of nitric oxide (NO) which then reacts with ozone (O3) to form nitrogen dioxide (NO2) (Gardner and Dorling 1999).

The hypergolic interactions between monomethylhydrazine (MMH, CH3NHNH2) and various forms of nitric acid were studied by several different experimental techniques.

There are 3 protein forms of nitric oxide synthase (NOS): neuronal NOS, endothelial NOS (eNOS), and inducible NOS (iNOS).

However, studies have reported increased expression of the endothelial (eNOS) and inducible (iNOS) forms of nitric oxide synthase in human [ 58] and murine [ 59] breast cancer tissue.

83 Blocking neuronal nitric oxide synthase activity inhibits apoptosis in neurons, and blocking all forms of nitric oxide synthase inhibits apoptosis in neurons and astrocytes, which suggests that nitric oxide is the key mediator of tat-induced apoptosis.

Physiologically, the expression of these cell adhesion molecules also appears to be limited by the formation of NO through either constituitive, or inducible forms of nitric oxide synthase (eNOS, iNOS).