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However, data on the frequency, impact, and predisposing factors for this pathology are still rare.
A B-profile can represent cardiogenic pulmonary edema [20 22] but is not specific for this pathology.
Inability to correctly identify HAGL lesions on preoperative imaging or during intra-operative evaluation will inevitably lead to lack of proper treatment for this pathology.
There is a lack of papers with a significant number of patients that were surgically treated for this pathology in the literature due to its rarity.
Interestingly, this NF2 KO phenotype was completely prevented in mice that were also haplo-deficient for Yap, indicating that augmented Yap activity is responsible for this pathology [99].
This article focuses on the particular context of the contamination epileptic signals (interictal spikes) by muscle artifact, as EEG is a key diagnosis tool for this pathology.
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There seemed to be a critical threshold for the distance between B-scans in the 3D-cube scan for detection of this pathology.
Therefore, the lack of the GPI anchor might be important for development of this pathology.
In the last decades different methods have been developed for treatment of this pathology.
Pharmacological compounds regulating immune responses are suitable candidates for prevention/treatment of this pathology.
Aggrecanases are potential therapeutic targets for the treatment of this pathology.
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