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In this study, we designed a set of experiments to test the hypothesis that GABA is required in non-neural cell types for the normal development and morphogenesis of the palate and retraction of the umbilical hernia.
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However, much remains to be learned about how apoptosis acts during normal development to actively induce tissue modification, and to what extent signals involved in injury and stress paradigms also contribute to normal development and morphogenesis.
In this review, we discuss the application of 3D culture models for studying branching morphogenesis of the mammary gland and the mammalian lung in the context of normal development and disease.
These results suggest that IKKβ is dispensable for normal development, morphogenesis and differentiation of the corneal epithelium.
Proliferation of smooth muscle cells is an important component of pulmonary arterial morphogenesis, both during normal development and pathologic remodeling.
Regeneration could depend on reiterative expression of genes that function in patterning, morphogenesis, and metabolism during normal development and homeostasis.
Vitamin A and its derivatives (retinoids) are crucial for the development, maintenance and morphogenesis of the central nervous system (CNS).
p63 is critical for maintaining epithelial development and morphogenesis (Senoo et al., 2007).
Moreover, p63 expression is critical for the maintenance of the progenitor cell population necessary for development and morphogenesis.
Proper ossification of the cranial bones and subsequent fusion of the cranial sutures provide protection for the central nervous system while allowing for expansion of the brain throughout childhood.[1] Deviation from the normal complex process of craniofacial development and morphogenesis results in various pathological congenital malformations.
Our work suggests that it is the changes in adhesion mediated by FasIII that are important for development and morphogenesis.
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