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A recent study suggested a role for the complement membrane attack complex in experimental models of osteoarthritis.
Recent evidence suggests an important role for the complement system in the etiology of age-related macular degeneration (AMD).
To implement the practical application of S-layer characteristic, the examination on more bacterial genotypes is required for the complement catalog.
Tissue salvage can be achieved by depletion of complement components, thus making evident a contributory role for the complement cascade in ischemia/reperfusion injury.
If ℐ is a proper non-trivial ideal, then the family of sets F ( I ) = { M ⊂ X : M c ∈ I } is a filter on X (where c stands for the complement).
Zymosan A prepared as described earlier (Craddock et al. 1977) was used at a concentration of 4 mg/mL in the serum as a positive control for the complement system activation.
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The amount of intracellular parasites decreased in the presence of normal human serum when compared to invasion in the absence of serum for the complement-sensitive strains (Figure 4B), and a slight difference in invasion rates was detected between them.
For the complementing observation A8_ΔΦ, the differences between simulation and experiment were more pronounced.
Previous studies have provided inconsistent results on whether variants in the MBL2 gene, coding for the complement-activating mannan-binding lectin (MBL) protein, associate with rheumatoid arthritis (RA).
Recent studies have reported that mice deficient for the complement-inhibitory protein CD59 show enhanced susceptibility to antigen-induced arthritis and reported that statins have anti-inflammatory effects in RA.
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