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We infer that the methylation of InAs to DMA is protective against respiratory effects of As, because those with a higher ratio of DMA/MMA appeared to be at decreased risk for lung dysfunction.
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Therefore, that study suggests that atelectasis is the most likely cause of lung dysfunction in more than half of patients who fulfill clinical criteria for acute lung injury after trauma.
Thus, objective assessment of lung dysfunction would be of benefit.
Similarly, both fluid overload and acute respiratory distress syndrome/acute lung injury (ALI) were chosen as justification for the insertion of six catheters (four PiCCO and two PAC), with the catheter used to help diagnose the cause of lung dysfunction.
In addition, numerous lung cancer survivors suffer from lung dysfunction, particularly patients with the lung surgical history [ 3].
Thus V/ Q imaging can detect early changes to the lung caused by cigarette smoke and can provide a non-invasive method for longitudinally studying lung dysfunction in pre-clinical models [ 76].
Contemporary animal and cell culture models for alcohol-induced lung dysfunction are discussed, with emphasis on the effect of alcohol on transepithelial transport processes, namely, epithelial sodium channel activity (ENaC).
Fig. 3 Schema showing novel mechanism for SHS-induced pediatric lung dysfunction.
These proinflammatory substances could be valuable predicting biomarkers for lung injury and/or lung dysfunction.
Thus interventions that can reduce pulmonary exacerbations are likely to be important for preventing future adult lung dysfunction [ 48] in addition to reducing the economic and social costs associated with each episode [ 49].
Furthermore, our outcome measures (change in SOFA-R and dynamic pulmonary compliance) do not fully represent change in lung dysfunction or the potential for pulmonary oxygen toxicity.
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CEO of Professional Science Editing for Scientists @ prosciediting.com