Microglial cell movement data (Figs. 5 7) was compared for significance using mean ± SEM of cell counts transformed via √(n+1) because of low cell counts [43].
Furthermore, these analyses were performed only for CVD and for men and women combined, due to low cell count.
Over five months 146 women gave their consent for an HPV test, of these six were excluded for previous LLETZ, two for an inadequate biopsy specimen and five for a low cell count in the liquid based cytology, which does not allow an additional HPV test.
Median survival time was 35.1 ± 10.8 months for the high cell count group and 20.3 ± 3.0 months for the low cell count group, respectively.
In our study, neither CD4 T-cell count (even for patient with very low cell count as <50/mm3) nor anteriority of HIV diagnosis, viral load, HAART at admission, reason for admission, and immunovirological status were significantly associated with ICU mortality.
In our study, neither CD4 T-cell count (even for patient with very low cell count as <50/mm) nor anteriority of HIV diagnosis, viral load, HAART at admission, reason for admission, and immunovirological status were significantly associated with ICU mortality.
†No information was presented for the excluded population due to low cell counts.
We minimized data suppression issues by regional aggregation of cases, yet for some of the race/ethnicity categories, low cell counts, particularly during the reference period, and underreporting combined to mask some of the relevant RR calculations.
This procedure facilitates using different sampling frequencies for areas with very high and very low cell counts.
Low cell counts precluded gene expression studies.Cilengitide was well tolerated but had no detectable clinical activity.
Because of heterogeneity between the studies we have conducted stratified analyses for low CD4 cell count, HAART exposure and region.
