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HOX genes are important for human endometrial development and receptivity.
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ADM is also an autocrine growth factor for human endometrial endothelial cells and is clearly involved in endometrial angiogenesis (Nikitenko et al, 2000).
Coudyzer, P. et al. Hypoxia is not required for human endometrial breakdown or repair in a xenograft model of menstruation.
The pathophysiology and the mechanisms of tissue repair in the endometrium are moreover poorly understood despite several theories on the source of cells for human endometrial regeneration [2].
Either way, these data provide further support for a functional role for AM in human endometrial repair.
Study Design: With use of a newly developed two-dimensional gel electrophoresis assay, we scanned 19 high-risk DNA fragments for alterations in human endometrial hyperplasias and adenocarcinomas.
The development of human endometrial carcinoma (HEC) is a complex pathologic process involves several oncogenes and tumor suppressor genes.
It was implicated in human endometrial receptivity, folliculogenesis as well as growth, development, and regression of the corpus luteum in higher mammals [ 87– 87].
Our findings represent a new concept regarding the mechanism of GnRH-II-induced cell motility in endometrial cancer cells and suggest the possibility of exploring GnRH-II as a potential therapeutic target for the treatment of human endometrial cancer.
Our findings provide a new insight regarding the mechanism of GnRH-II-induced cell motility in endometrial cancer and suggest the possibility of exploring GnRH-II as a potential therapeutic molecular target for the treatment of human endometrial cancer.
To develop a protocol for cryopreservation and recovery of human endometrial epithelial cells (eECs) retaining molecular and functional characteristics of endometrial epithelium in vivo.
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