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For example, the expression of the α-factor protease Bar1 improves performance at high pheromone concentrations.
For example, the expression of the atonal TF in Drosophila melanogaster (Dmel) in different tissues is regulated by discrete CRMs, some of which are also autoregulatory [2] [4].
For example, the expression of the peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) in humans and rodents is temporarily elevated after each exercise bout [16], [17], [18], and exhibits a persistent basal elevation after chronic endurance training [19], [20], [21] while maintaining additional inducibility with physical activity [22], [23].
This can be based on experimental data, for example the expression of the genes that the edge connects.
For example, the expression of the KCNN3 gene decreased in GCs during early development whereas its expression increased in oocytes.
For example, the expression of the Ag-TEP1 gene is upregulated after immune challenge and, upon activation, it has been shown to promote phagocytosis [ 37].
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For example the expression of carbamoylases is reported to be inducible (Mei et al. 2008).
Examples of regulatory mechanisms for the expression of the catabolic genes are given.
Dioxin, for example, induces expression of the activating enzyme CYP1B1 [75] 75] (Fig. 2).
For example, the facial expression module best categorized the happy expression images, whereas the speech module determined the speech of sad emotion with the best accuracy.
For example, transgenic expression of Sir2 in the fly brain can confer lifespan extension [11].
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