For example, expression of the non-coding, maternally expressed gene Meg3 has been shown to be lost in many kinds of primary human tumors and tumor cell lines.
For example, expression level and gene length showed opposite correlation patterns in highly and lowly expressed genes, as was previously shown (Carmel and Koonin 2009).
Consider for example expression (fst({ True},o)) (cf., Sect. 2), where (o) has any type with non-empty constraints.
For example, expression of scpA was decreased in a biofilm but elevated in a sarA mutant.
For example, expression of TGFβ1 is increased in stromal cells as they undergo decidualization [20], [21].
For example, expression levels that are too low for effective competition with wild-type proteins.
For example, expression of Sema3E in prostate cancers and metastatic melanoma inhibits adhesion and motility of cancer cells [38], [39].
For example expression traits associated with cigarette smoking were more strongly associated with carotid atherosclerosis than smoking itself.
For example, expression of full-length CFTR protein at the apical cell membrane was observed in cystic fibrosis patients [17].
For example, expression of the replication-related proteins encoded by CPMV RNA-1 is known to induce a massive proliferation of endoplasmic reticulum (ER -derived mER -derived0]–[12].
For example, expression signatures that identify sub-classes of cancers with aggressive characteristics or cancers resistant to commonly used therapies represent therapeutic opportunities [10], [11], [12], [13], [14].
