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By associating cell-type-specific expression programs with key disease genes, we establish a new cellular narrative for airways disease.
Twenty four hours after the last exposure, we analyzed the mice for airways hyperresponsiveness (AHR).
Mice were exposed daily for 10 consecutive days, and 24 hours after the final HDM exposure, analyzed for airways hyperresponsiveness, then sacrificed, samples collected for histology, and RNA obtained from lungs as detailed below.
Therefore, we only pooled results for airways ≥ fifth generation.
Having evidence for airways obstruction (i.e., a low FEV1 to FVC ratio) was not apparently associated with current occupation.
PvdD production was shown to be required for airways bacterial colonization in rat, lethal virulence in burned and immunosuppressed mouse models [ 35, 36].
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Interferon alfa-2a therapy for airway hemangiomas.
Roy, M. G. et al. Muc5b is required for airway defence.
There were no readmissions for airway problems.
Laryngeal sensation is critical for airway protection and safe swallowing.
Supraglottic airway devices are designed for airway management during anaesthesia.
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