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Supporting AtHB8, AlHB8, and MgHB7 within the HB8 clade is evidence for a duplication event resulting in the HB8 and CNA clades, as previously reported (Prigge and Clark, 2006).
Each clade in the phylogenetic tree stands for a duplication group in our analysis.
To our knowledge no phenotype has been demonstrated for a duplication of this gene, but clinical relevance cannot be excluded.
In our study, we did not find evidence for a duplication event in the case of this gene.
In contrast, Tree-10 had much less support for a duplication on this branch and Tree-10 had even less.
There is strong support for a duplication that gave rise to the SMC1/4 lineage, but weaker support for the SMC2/3 duplication.
Similar(30)
First, we determined the frequency of duplicated segments for (a) duplications within the complex region, (b) duplications between the complex region and other regions in the same chromosome, and (c) duplications between the complex region and other regions in different chromosomes.
This scenario is supported by the analysis of contemporary tRNAsGly demonstrating a highly conserved anticodon loop CCA sequence [ 34] and previous statistical analyses of tRNA molecules by Di Giulio and colleagues arguing for a duplication-ligation origin of tRNA [ 31- 33, 62].
For instance, a duplication of the agouti signaling protein gene (ASIP) in sheep results in a different pigmentation [ 17].
For example, a duplication between the α2 and α1 genes in human is reapplied, as the dotted circles and arc in figure 4 A and B show.
For example, a duplication of a set of FGF genes in Rhodesian and Thai Ridgebacks leads to the breeds characteristic dorsal hair ridge [ 19].
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