Sentence examples for folds control from inspiring English sources

The eastern Cordillera of the Bolivian Andes is an extremely wide fold and thrust belt, but only along the eastern third of the cordillera do simple parallel folds control the topography.

Growth inhibition was calculated as fold control.

Phosphorylation of AKT1 (1.28 vs. 0.94 fold control) and SAPK/JNK (1.77 vs 1.34 fold control) was reduced, although significance levels varied between 0.05 and 0.15.

Surprisingly, ERK1/2 was significantly up-regulated (1.23 vs 1.50 fold control, p<0.05).

The VEGF control (262 p M) observed a ∼2.4-fold increase over control, which was suppressed back to ∼1.5-fold control by 1  μ M SU1498.

Four hours pre-incubation with papain lead to a significantly reduced phosphorylation of MEK1 and p38-MAPK after activation with VEGF (2.13 vs 1.58 fold control resp. 1.32 to 1.14 fold control).

Muscle samples were first diluted 60-fold in PBS, subsequent dilutions and the calibration curves were done in 60-fold control muscle in PBS.

However, co-transfection with both sip16 and Sall2 generated a G2/M population of 9.35%, or 1.2-fold control level, a significant decrease from the 1.5-fold sip16 alone caused by co-transfected Sall2, but p16 knockdown dramatically abolished most Sall2-induced G2/M reduction (from 0.57-fold of control to 1.2-fold of control) indicating that p16 is critical in Sall2-induced cell cycle inhibition.

At 72 h, αKG increased 50%>48-h>48-hels, pyruvate was further depleted and lactate increased to more than 3-fold control levels (Fig. 7H).

DACH1 706aa- and 709aa-overexpressed H295R cells also increased the canonical Wnt signaling activity to 3.2 and 3.9-fold control vector, respectively (P=1.0×10−13 and 8.7×10−14, respectively; Figure 4C).

BLA lesion suppressed proliferation ipsilateral to the lesion by approximately 1.5-fold (1.66-fold control, 1.44-fold immob), as we have previously reported, but did not block the stress-induced increase in proliferation.