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The changes can be thought of in terms of a scale where the normal membrane is assigned a value of zero, a fluid membrane in minus numbers and a rigid one in plus numbers.
The motions of the fluid, membrane and permeation flux are coupled monolithically and time-advanced fully-implicitly.
We calculate the influence of a flexible network of long-chain proteins, which is anchored to a fluid membrane, on protein diffusion in this membrane.
In the same manner, according to SEM EDS results (Fig. 5), the decompositions in H3O+ medium at low [H3O+] are described by the unreacted core model without the formation of the solid product layer, even if the results in Fig. 8 point to an intermediate behavior between controlling stages (diffusion in the ash layer and diffusion in the fluid membrane).
The observations are compatible with a higher V1 off rate in a more fluid membrane.
The lipid raft concept posits that these are well-organized platforms of lipids and proteins floating in the fluid membrane.
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Vesicles are locally-inextensible fluid membranes that can sustain bending.
These mechanisms lead to less fluid membranes, more hydrophobic MA and increased cell surface hydrophobicity.
The eukaryotic endosomal trafficking is essential for the internalization and trafficking of macromolecules, fluid, membranes, and membrane proteins.
Saturated FAs in the raft sphingolipids and phospholipids are closely packed with cholesterol resulting in more ordered and less fluid membranes than the rest of the PM.
Here, we show that the incorporation of physiologically relevant contents of cholesterol in model fluid membranes produces a fourfold increase in the membrane bending modulus.
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