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(B) Evolution of growth parameters with inherited growth and fixed maturation parameters (M1− M18 = 400 mm) including density-independent growth.
For baseline simulations with fixed maturation or growth parameters, we set M1– M18 to 400 mm and X1– X12 to 0.5, respectively.
This interaction between growth and maturation parameters was qualitatively similar across sexes to fixed maturation and growth parameters.
We also explore the effects of the magnitude of fixed maturation parameters on inheritance of growth, and vice versa, by running simulations with M1– M18 set at 300 or 500 mm and X1– X12 set at 0.3 or 0.7.
Evolution of (A) maturation parameters for males (left) and females (right) in baseline simulations with inherited maturation and fixed growth parameters (X1− X12 = 0.5) and including density-independent growth.
Similarly, when growth parameters are fixed and maturation parameters are heritable, fixed growth parameters exert selection (albeit relatively weak) on genetically determined maturation length thresholds.
It is generally thought that the KIR receptors are acquired by a stochastic mechanism, currently poorly understood, which operates exclusively during NK cell development, and that the repertoire is fixed after maturation.
These baseline simulations can be grouped into four general categories: (i) maturation and growth parameters are fixed, (ii) only maturation parameters are heritable, (iii) only growth parameters are heritable, and (iv) both growth and maturation parameters are heritable.
Our simulations suggest that when size at maturation is fixed and growth rates are partially genetically determined, the maturation length threshold has potentially strong influence on the genetic component of growth.
However, evolution of growth parameters was influenced by maturation fixed at certain levels and vice versa (Fig. 4).
The specimen of Stomopneustes variolaris (Lamarck, 1816) shown here was fixed before gonad maturation.
