First, we observed that gene expression variation of selected genes could precede clinical observable manifestations.
First, we observed that there was no apparent change in total ubiquitinated protein levels following PHGDH KD (Fig. S2B).
First, we observed that, as a whole, genes in the positive reference sets were significantly differentially expressed between tumour and normal samples.
First, we observed that the mechanical properties of the EVL basal surface (the substrate for DCL spreading) were distinct at EVL cell borders compared to non-border regions.
First, we observed that DSF/Cu induced significant cytotoxicity and caspase-dependent apoptosis in NSCLC cells, accompanied by the formation of vacuoles in the cytoplasm.
First, we observed that the perimysium is made of a highly ordered network of collagen fibers, binding the myofibers from tendon to tendon.
First, we observed that tumor growth of PyMT-mCh-OVA, a variant of PyMT-B6 expressing antigenic mCherry and ovalbumin, was restrained in wild-type, but not in BATF3−/− mice (Fig. 3e).
First, we observed that apoptosis of MIN6 cells, induced by Alloxan as well as IL-1β, requires activation of the AMPK pathway, and also that CNTF protective effects are dependent on downregulation of AMPK.
First, we observed that, like the rat, LiCl activates preproglucagon neurons in wild-type mice.
First, we observed that the KL-divergence values are always less than 0.05.
First, we observed that the morphology is different from that of the Se/Te NWs with a straight morphology reported previously [23].
