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Our data, for the first time, demonstrate the importance of intracellular thiols in sequestering toxic reactive aldehyde products of lipid peroxidation and polyamine metabolism.
These findings for the first time demonstrate that TNFAIP8 is one of critical components of a signal transduction pathway that links mesangial cell proliferation to diabetic renal injury.
Our data, for the first time, demonstrate that hypoxia significantly enhances ROS production, measured by the standard ROS indicator dichlorodihydrofluorescein/diacetate, in isolated mitochondria from PASMCs.
We achieve a record fracture toughness enhancement and, for the first time, demonstrate a coherent quantitative correlation between the fracture toughness and the surface roughness.
Obtained results for the first time demonstrate a coalescence determined increase in bubble Sauter diameter d32 from 187 to 211 µm with increasing stirring intensities.
Here, we for the first time demonstrate the use of GQD and sulfonated GQDs (SGQDs) as quasi-homogenous catalysts for chemo-catalysis.
The results, for the first time, demonstrate that ECE can be tailored and enhanced through nanocomposite approach in the ferroelectric polymers.
Here, we uniquely combine these two growth strengths (spinodal decomposition and strain field) and, for the first time, demonstrate a general and well-repeatable method to produce coherent and self-organized magnetic nanostructures with superior magnetoresistance in the MnGe system.
The present results support and extend the enhancing effects of crocins on memory and, then, to our knowledge, for the first time, demonstrate its implication in the mechanisms underlying recognition and spatial memory.
We for the first time demonstrate that these mesopore system could be loaded much more deposits in them, and confine the size of lead deposits to be in nano scale by their local effect, ensuring a much more remarkable suppression of hydrogen evolution and better reversibility of Pb/PbSO4.
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These studies for the first time demonstrate that, like BM-MSC, neural crest-derived stem cells tolerate EWS-FLI1.
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