Finally, we inspected whether there were differences among the coloration groups in the values of the RSM.
Finally, we inspected the distributions of childhood cognitive test scores (as density plots) in order to ascertain whether any effects were due to a subgroup of individuals with high or low scores, or whether they might be due to shifts in the whole distribution of scores between groups with and without symptoms.
Finally, we manually inspected and extended the 4D-CHAINS results to establish the maximum number of highly accurate assignments for all 13C 1H correlations that can be observed in our 4D spectra (>98%), as a "best-effort" resonance list requiring a modest time investment by a trained user.
Finally, we carefully inspected irradiated hippocampal sections for evidence of inflammation, apoptosis, or necrosis using hematoxylin and eosin (H & E) and Hoechst stains.
Finally, we systematically inspected the preprocessing stream used in all five samples in order to identify any potential steps that might introduce systematic artifact correlated with trial-by-trial RT differences.
Finally, we manually inspected the results to confirm that they showed evidence for being putative transcripts.
Finally, we visually inspected CNV regions identified by the automatic procedures to exclude aberrant CNV segments (in a CNV region, only one conspicuously long segment with almost the same signal intensity as that of two copies).
Finally, we manually inspected the results in.b ed format on each chromosome arm of D. melanogaster to look for cluster of sequences with a higher identity than expected using the integrative genome viewer software (Thorvaldsdóttir et al. 2013).
Finally, we graphically inspected the 96 genes that met the above criteria and manually selected genes that were robustly different between the alternate met1 genotypes at several time points.
