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Finally, we identified that p38γ MAPK overexpression led to marked cell cycle arrest in G2/M phase.
Finally, we identified that p38 MAPK and ERK1/2 signaling pathways regulated the internalization of Aβ1 42 via LRP1.
Finally, we identified that Gli1 and Gli2 exhibited different functions in the regulation of p63 expression or proliferation of p63+ cells in Kras-AR driven tumors.
Finally, we identified that eukaryotic translation elongation factor eEF1A1, one of the most abundant protein synthesis factors [24], might be a novel target for cilostazol.
Finally, we identified that ChAT- and acetylcholinesterase-expressing axons are present in heterotopia.
Finally, we identified that UHRF1 is regulated by miR-9 in CRC by dual luciferase reporter assay.
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Finally, we identify that the photoluminescence (PL) of the nanowires has direct relations with the diameter and the crystal quality of InP nanowires at different growing stage changes.
Finally, we identified genes that were present in human, chimp, and yeast but that varied in their presence and absence in the "intermediate" lineages between human/chimp and yeast (fig. 3 A ).
Finally, we identified factors that may have a substantial influence on performance of particular methods.
Finally, we identified loci that explain a significant amount of heritability across multiple traits.
Finally, we identified compounds that potently dissolved vesicle-bound α-synuclein oligomers into monomers, leaving the lipid vesicles intact.
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