Finally, we cannot exclude a 'healthy worker' effect of the final sample with full data (although this effect would point to a conservative estimate of the observed effect), and we cannot solve the methodological problem of reporting bias as both types of variables, the measure of stressful experience at work and the measures of mental health, were based on self-reported data.
Finally, we cannot exclude that different results could have been found if we had measured EtCO2 after stopping NIV treatment.
Finally, we cannot exclude the possibility that some individuals in whom the device was effective did not purchase it for financial reasons, which would have led to an overestimation of the proportion of non-satisfied subjects.
Finally, we cannot exclude the possibility that next to mechanical ventilation, the procedures related to the instrumentation/ventilation (e.g. intubation, arterial cannulation) also induce inflammation to a certain extent.
Finally, we cannot exclude that differences in placebo arms between disorders may reflect differences in the probability of spontaneous improvement.
Finally, we cannot exclude that Val-145 is involved in binding of a yet unidentified domain on leptin receptor.
Finally, we cannot exclude that the OK371-Gal4 expressinneuronsurons is incomplete because it lacks promoter/enhancer elements in the construct.
Finally, we cannot exclude the possibility that Sgs1 plays a role in suppressing the segregation of non-exchange chromosomes and that this activity is dependent on an interaction with Mlh1.
Finally, we cannot exclude a short-term benefit from anticoagulation during the acute hospitalization phase post-MI as the number, and information on the timing, of stroke prior to discharge is limited.
Finally, we cannot exclude the possibility of residual confounding.
Finally, we cannot exclude the possibility of residual confounding and confounding by unmeasured variables.
