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It has been proved that, fiber interaction substantially intensifies stress concentrations within the interphase region, although imperfect adhesion relieves stresses.
Cell cellulose fiber interaction is the determinant signal in the cell biomaterial responses, isolated from other frequently present interferences such as protein and other chemical traces usually present in cell culture matrices.
Comparative studies of the various micromechanical theories/models with FEM/Strain energy method for the prediction of damping coefficients have shown consistency when both the effect of variable nature of stress and the fiber interaction is considered.
While these mechanisms of clonal selection for epigenetic silencing are consistent with our data, it does not necessarily imply any direct asbestos fiber interaction with the histone/DNA methylation machinery, but instead that the chronic inflammation response and/or accelerated tumor growth related to asbestos burden may select for cells capable of continued proliferation.
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It is thus conceivable that CTCF and components of the cohesin complex provide sufficient affinity between interacting CTSs to stabilize, perhaps even to immobilize, chromatin fiber interactions (Wallace and Felsenfeld 2007) resulting from proximal stochastic collisions (Fig. 1).
It is shown that the gate design and different levels of fiber interactions, due to different fiber concentrations, are responsible for these observations.
The presence of annulospiral wrappings (ASWs) and flower-spray endings (FSEs), both physiologically relevant morphologies in sensory neuron-intrafusal fiber interactions, were demonstrated and quantified using immunocytochemistry.
The flexural wave and projectile fiber interactions induce curvatures in the fiber significant enough to induce compressive fiber failure and fibrillation.
The shear anisotropy we report is consistent with prior anatomical observations of this tissue and appears to develop through separate contributions from the matrix, the collagen fibers, and collagen fiber interactions.
Studying single cell – single fiber interactions with the aid of confocal laser scanning microscopy (CLSM), scanning probe nanotomography (SPNT), and transmission electron microscopy (TEM), we found that cardiac cells actively interact with substrate nanofibers, but in different ways.
Since we previously found that PRV infection induces mast cell accumulation in the lamina propria [11], [12], [17], the location of C fibers within the bladder lamina propria raises the possibility that mast cell-C fiber interactions contribute to PRV-induced pelvic pain.
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