At admission, the girl was lethargic and inappetent with a fever of 38.3°C and oxygen saturation of 86%.
However, on the 3rd day of treatment, he developed sudden desaturation with resurgence of high fever, and required supplemental oxygen therapy.
Bernard et al. [ 10] reported that, in patients with sepsis, treatment with ibuprofen reduced levels of prostacylin and thromboxane and decreased fever, tachycardia, oxygen consumption, and lactic acidosis, but did not prevent the development of shock or acute respiratory distress syndrome, and did not improve survival.
Three weeks later, he had upper respiratory tract symptoms, low-grade fever, and protracted cough; blood oxygen saturation was low and respiratory distress had rapidly increased.
On day 15, he developed dyspnea, cough, mild hemoptysis, and oxygen desaturation without fever.
In volunteers, induction of fever and active external cooling increased oxygen consumption up to 40% and was associated with a significant increase in catecholamine levels [ 62].
She didn't have a fever, and her blood was getting enough oxygen.
Fever may have detrimental effects, especially in life-threatening illnesses, by increasing the metabolic rate, minute ventilation and oxygen consumption, and by adversely affecting neurological outcomes [ 5- 7].
And oxygen masks dropped.
