Sentence examples for fetal pathology from inspiring English sources

2. Optimal intrauterine growth is most likely to be achieved during pregnancies unaffected by any maternal or fetal pathology or exposures that can pathologically affect fetal growth, 3. The many determinants of fetal growth can be classified as having either a pathological or a non-pathological effect on growth.

He is also known for his writings on medical history (especially on leprosy in Scotland) and on fetal pathology and hermaphroditism.

Many animal models have been used for vaccine, drug testing, or studies on host or parasite factors that affect transmission or fetal pathology, but few works have directly tested fetal infection and damage rates along gestation.

As a consequence of maternal, placental or fetal pathology the fetus cannot fully exploit its growth potential [1].

Intrauterine growth restriction (IUGR) is a fetal pathology which leads to increased risk for certain neonatal complications.

It often is difficult to induce isolated single maternal perturbations related to the above complex phenomena and in vivo model approaches which dissect specific maternal-sided contributors to fetal pathology are needed.

Applications of these techniques are used to monitor the details of blood-flow patterns in the uterine arteries, to measure placental perfusion, and to characterize angiogenesis and vascular permeability, providing insight into placental and fetal pathologies.

In Iceland, Denmark and Norway, women with major fetal pathologies were excluded from the study.

We found that mothers who experienced active depression or anxiety in pregnancy were more likely to deliver SGA/IUGR infants, and our follow-up epigenetic analyses suggest that altered leptin signaling may be involved in the biologic mechanisms that cause these maternal and fetal pathologies to co-occur.

Nonetheless, the investigation of fetal pathologies such as T18 and T21 cannot be fully replicated in other organisms, and these results suggest that epigenetic changes between disease groups can be identified, as long as there is careful control for all confounding factors such as gestational age and consideration of the effects of tissue composition.

Ultrasonographic characteristics, associated syndromes, chromosomal anomalies, fetal cardiac pathology and life after birth were considered.