Sentence examples for fate decision making from inspiring English sources

Exit from the cell cycle is intimately coordinated with the cell fate decision making process in RPCs [2].

The experimentally controlled differentiation of pluripotent cells represents a good model system to understand the general process of fate decision making in development (Gadue et al., 2005; Murry and Keller, 2008).

The expression of these novel mouse genes in early embryos would fit well with recent data tying known polydactyl ZNF genes to cell fate decision making and early development (Quenneville et al. 2012; Santoni de Sio, Barde et al. 2012; Santoni de Sio, Massacand et al. 2012; Barde et al. 2013; Corsinotti et al. 2013; Schep and Adryan 2013).

In particular, we focus on the characterisation of biological information processing efficiency, examining cell-fate decision making processes, gene regulatory network reconstruction, and efficient signal transduction experimental design.

Given that both of these genes have been shown to be possible regulators of important pathways that are also at work in the developing retina, they make excellent candidates for regulators of the cell cycle exit and cell fate decision-making processes of RPCs.

Changes in the stiffness of the extracellular matrix can have a profound influence on the fate decisions made by stem cells [5], [6].

This suggests that a Tcf-dependent binary decision model may also apply to fate decisions made during vertebrate neuronal development.

Depending on the cell fate decisions made between isolation and transplantation, donor cells can contribute to the skin, brain, or eye of host embryos.

We know that the cell fate decisions made by retinal progenitor cells are governed by an intrinsic genetic program that determines their response to extrinsic cues from their environment.

This antagonism is often seen in the cell-fate decisions made by individual cells with Wnt signalling promoting the adoption of one cell fate and Notch another.

However, it remains possible that Notch signaling may play some role in cholangiocyte differentiation, since none of the three mouse models analyzed (Hes1-null mice, Jag1dDSL/+ Notch2del1/+ mice, and Notch2-cko mice) are likely to be entirely deficient in Notch signaling when the cholangiocyte-hepatocyte cell fate decision is made.