Sentence examples for fate activities from inspiring English sources

Analysis of up-regulated targets indicates that under cold stress there are 171 up-regulated target genes of BHLH, several of which are connected to metabolism, transport activities, stress response and cell fate activities (p ≤ 0.05) (table 4).

This plant model, therefore, provides a unique opportunity to dissect cell division and cell fate activities of Rb and other conserved proteins during programming and reprogramming and is a powerful comparative system for future discoveries of fundamental regulatory mechanisms of stem cell initiation, maintenance and termination.

Together with the many associated genes that are known to regulate cell fate, activity and morphology, this seems to underline the importance of neural tissue plasticity in the appropriate expression of estrous behavior.

We hypothesized that earthworms modify the abundance of indigenous soil bacteria and the fate and activity of atrazine-degraders in the soil they engineer by bioturbation.

Therefore, identifying genes that regulate APP steady-state levels, APP cleavage, and the fate and activity of AICD are likely to further our understanding of AD pathogenesis.

Proteolytic cleavage plays an essential role in the control of numerous biological processes, including protein localization, fate and activity as well as the processing of cellular information.

Thus, the middle layer changed fate and activity more times in male-sterile than in male-fertile anthers, converting from an initial function of transporting materials towards the tapetum to secretory activity, thus assuming the function of the degenerated tapetum, and finally to a mechanical function, possibly to strengthen the residual anther wall.

There are several mechanisms by which a cell may achieve this, and they include altered transcription, translation, protein trafficking, post-translational modifications (PTMs) and, in particular, selective and limited cleavage at the plasma membrane, which controls the fate and activity of various cell surface proteins.

TSCM have been demonstrated to persist, while preserving their precursor potential in bone marrow transplanted patients for up to 12 years after infusion of gene-corrected hematopoietic stem cells, or mature lymphocytes that were tracked concerning their fate and activity [ 24].

Data from spinal cord injury models demonstrate a link between mTOR activity and cell fate, with activation of PI3K signaling stimulating differentiation of preoligodendrocytes to astrocytes.

We propose that the Pou3f1-dependent repression of the BMP and Wnt signaling pathways and the activation of intrinsic neural lineage genes together are involved in the neural fate-promoting activity of Pou3f1.