Sentence examples for failures to complement from inspiring English sources

It is expected that if these conditions are satisfied, failures to complement due to the presence of other QTL outside the region in question can be ruled out.

Although our results with Df(1 ED6906 and Df(1 C128 meet the requirements to be considered allelic failures to complement, these can also be interpreted as an epistatic failure to complement due to interactions of these deficiencies with other loci that affect CCRT residing elsewhere on the X or in the other two major chromosomes.

To test whether epistatic interactions between genes on the wild-derived chromosomes and the mthR3 background were responsible for the failures to complement, we used an F-statistic to examine the equivalency of variances in both the mth wild-type and mutant backgrounds [31].

Of the 16 candidate regions and genes tested, Df 2L c17, Df 3L Ly, Df 3L AC1 and Df 3R e-BS2 showed significant failure to complement wild-type alleles in both sexes, and an Alcohol dehydrogenase mutant failed to complement in females.

In vivo expression of the full-length and N-truncated DHODHs from engineered transgenes revealed that the truncated proteins could not support normal de novo pyrimidine biosynthesis during development of the fly (i.e., failure to complement dhod-null mutations), apparently due to instability of the truncated proteins.

Values for their specific activities range from 40% of that displayed by wild-type enzyme to complete inactivation as evidenced by failure to complement an ilvA deletion strain to isoleucine prototrophy.

Failure to complement resulted in siliques that continued to segregate for normal and mutant seeds.

Failure to complement (xen72−3) might be due either to loss of the deleted region or to disruption of gene function by the presence of the P-element.

Thus, failure to complement l(2 gl does not correlate with suppression of TPE and is not diagnostic for a tip deficiency.

The following stocks were generated by standard meiotic recombination and isolated on the basis of their failure to complement other alleles and/or sequencing to verify the molecular lesion: NcadΔ14; elmbcD11.2spg242, mbcD11.2.

Nor does the failure to complement appear to be determined by epistatic interaction between the functional lab-derived wild-type allele and the wild-derived lines, as the F ratio tests were nonsignificant for all comparisons.